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These drugs, including trihexyphenidyl and benztropine, act by blocking muscarinic receptors, reducing the overactivity of cholinergic neurons that contribute to PD symptoms

A single molecular entity simultaneously activates both the GLP-1 receptor and the amylin receptor (calcitonin receptor complex), combining two complementary satiety and metabolic pathways: GLP-1 receptor activation : Glucose-dependent insulin secretion, glucagon suppression, delayed gastric emptying, and central appetite reduction through hypothalamic signaling Amylin receptor activation : Additional satiety signaling through the area postrema, complementary gastric emptying delay, and glucagon suppression through a distinct mechanism The unimolecular design means both receptor activations occur at a fixed ratio determined by the molecular structure

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10.1002/jcp.29391 42 LiS.HeY.ChenK.SunJ.ZhangL.HeY.et al (2021)